Increasing aromatase activity increases conversion of androgen to estrogen.
Estrogens can have an inhibitory effect on prostate cancer.
That is 'good,' so the answer actually is 'yes.'
It is not clear that doing this outside dealing with a cancer is rational because evidence suggests that prostate tissue can alter its relationship with androgens and testerosterone over the course of a man's life. For example, the western diet, characterized by high fat consumption, predisposes men to benign prostatic enlargement (BPH) while a diet rich in flavonoids and lignans, containing phyto-estrogens, which inhibit aromatase, lowers this risk.
Although the data would suggest that in the medical treatment of BPH, anti-estrogens or aromatase inhibitors could be used, trails of these have been disappointing. (1)
WHAT WE CAN GET OUT OF THIS
a. In essence, an enlarged prostate is sensitive to different hormones than a normal or cancerous one.
b. In a young man with a healthy prostate aromatase inhibition is probably a good idea. Bring on the flaxseed oil and chamomile tea!
c. It has no effect in men with benign prostate enlargement *, and;
d. The opposite may in fact be true for certain cases of prostate cancer. Here one could almost make the case that aromatase activity could be encouraged. Bring on the licorice!
1 Arch Androl 2000 May-Jun;44(3):213-20 Role of estrogens in human benign prostatic hyperplasia. Sciarra F, Toscano V
* By the way, there is an interesting connection here to our previous discussion of stinging nettle root (North American's UDA Plus), which I previously posted about as a potential anti-candida agent. In Germany, nettle root has been used as a treatment for enlargement of the prostate gland for decades. A metabolite of testosterone called dihydrotestosterone (DHT) stimulates prostate growth, leading to enlargement. Nettle root inhibits the binding of DHT to attachment sites on the prostate membrane.