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The Blood Type Diet Archives Volume 13

Soy, Type A, and Final Vocabularies

Posted By: Peter D'Adamo
Date: Wednesday, 26 January 2000, at 5:16 p.m.

In Response To: Soy Symposium link--Dr. D--QUESTIONS (Marilyn Lloyd)

Some points to remember, or at least ponder:

1. All estrogens stimulate estrogen receptor (ER) positive cells, including Tamoxifen, which is given clinically to inhibit these very same cells. Tamoxifen, like soy phytoestrogens, are 'competitive' with regular estrogen for the ER; they are just not a powerful a stimulant, and in sum total, because of this, actually lower estogenic activity.

2. Soy is a potent inhibitor of angiogenesis (the ability of a growing embryo or cancer cell to develop a collateral blood supply). As we will discover in LR4YT, angiogenesis is a common corollary with type A and malignancy, having to do with the ability of the type A antigen to fit the receptor for Epidermal Growth Factor (growth factors are hormone like substances which increase the metabolic rate of cells; many cancers hae higher numbers of growth factor receptors than otherwise normal cells) and a class of tissue lectins call 'integrins.' Because of this, 'anti-angiogensis' in a type A cancer patient is a wise strategy.

3. Soy bean lectin agglutinates virtually all breast cancer cell lines, and has additional effects with regard to enhancing the ability of a chemical in the blood stream called 'complement.' One type of complement stimulated by soy lectin helps clear 'immune complexes', the junk that results from and antibody interacting with an antigen. Immune complexes in general bad for your body's defenses, typically because they cause unneeded activation.

4. Soy beans doe slightly inhibit tyrosine kinase (TK), but I doubt that it is the 'smoking gun' of 'soy-induced brain atrophy' if in fact it is soy actually doing it in the first place. TK is an intracellular enzyme involved in energy production, inhibited by lots of other foods, many of them lectin containing. By the way, the low-bad lectin blood type specific diet will in general enhance tyrosine kinase activity by virtue of balancing polyamines (a class of chemicals involved in growth).

Again, we must go back to the basics of what the blood type paradigm challenges us to never forget: The diet for the tailor may not be the best diet for the blacksmith.

I've been reading the work of the philosopher Richard Rorthy. Very interesting. In 'Contigency, Irony and Solidarity' he makes the point that in many cultures the evolution is towards what he calls 'A final vocabulary' where no new words can be added. In these groups, this leads to greater complacency (bad) and security (probably good) because things are accepted by virtue of their ability to appeal to 'common sense.' The shortcoming of a 'final vocabulary' is that you have absolutely no ability to appreciate the ironic; and it is in the appreciation of things tha are ironic that we can grow conceptually. It will be years before researchers move away from 'final vocabulary' in nutrition, mainly because they believe they act with 'common sense.' Because of this, the irony of the blood type paradigm, such as a meat eating O lowering their cholesterol, is lost on them.

In short, we must be comfortable with our paradigm, and appreciate the irony it often provides us with. The alternative otherwise is 'common sense.'

Phoooey on that.

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